By Sharma S Prabhakar
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Additional resources for An update on glomerulopathies : clinical and treatment aspects
Pamidronate b. Lithium c. Interferon –alpha d. Heroin e. Sirolimus 3. Secondary FSGS mediated by adaptive structural-functional responses a. Reduced renal mass Unilateral renal agenesis Oligomeganephronia Renal dysplasia Reflux nephropathy Sequela to cortical necrosis Surgical renal ablation Chronic allograft nephropathy Any advanced renal disease with reduction in functioning nephrons b. Initially normal renal mass Obesity Hypertension Atheroembolic or other acute vaso-occlusive processes Cyanotic congenital heart disease Sickle cell anemia Anabolic steroids 4.
Periodic acid methenamine silver, 20X). (B-Right) Patchy effacement of foot processes is present. No immune complex-type deposits are seen along the GBM. (Electron Microscopy). Perihilar Variant of Focal Segmental Glomerulosclerosis : This variant is defined as perihilar hyalinosis and sclerosis (Fig 2. A & B) which involves more than 50% of glomeruli with segmental lesions. This category requires that the cellular, tip, and collapsing variants be excluded. Podocyte hyperplasia is uncommon. Fig.
Idiopathic Membranous Nephropathy: Outline and Rationaleof a Treatment Strategy. American Journal of Kidney Diseases, Vol 46, No 6 (December), 2005: pp 1012-1029. Dusso B, Morange S, Burtey S. American Journal of Kidney Diseases, Vol 52, No 4 (October), 2008: pp 699-705 Ehrenreich T, Churg J. Pathology of membranous nephropathy. Pathol Ann 1968;3:145. Ehrenreich T, Churg J. Pathology of membranous nephropathy. Pathol Ann 1968;3:145. Kidney International (2008) 73, 117–125 Floccari VF,Cosentini M.
An update on glomerulopathies : clinical and treatment aspects by Sharma S Prabhakar